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Their canonical mechanism is the competitive inhibition of SERT on presynaptic terminals, reducing serotonin reuptake and thereby increasing extracellular serotonin concentrations in the synaptic cleft [23]

Most studies reported diabetic neuropathy as one of the endpoints, rather than classifying it into peripheral neuropathy and autonomic neuropathy

These effects are often the result of slowed gastric emptyingthe very mechanism that increases satiety and reduces food intake

For the real-time internalization assay, the N-terminally SNAP-tagged GIPR was synthesized by Cisbio and inserted into pcDNA3.1

The regulatory requirements differ significantly

doi: 10.1186/s12902-025-01996-z