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The migration and tube formation ability of HUVECs are observed to be enhanced significantly after addition LRG1, whereas the load-induced hypertrophic scar formation resulted from pathological angiogenesis is ameliorated after depletion of LRG1 (Zhang et al., 2019)

Pourmasoumi M, Hadi A, Marx W, Najafgholizadeh A, Kaur S, Sahebkar A

doi: 10.1021/jf0519055

H.GlynnJ.HanM

Yet, the startling reality is that metabolic and nutritional factors, many of which are modifiable, are now recognized as primary drivers of its pathogenesis, contributing to a global burden that afflicts over 300 million people (1)

Inactivation of tumor suppressor genes, p53 and Rb1, in plasma cell dyscrasias