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Antioxidant therapeutic strategies in neurodegenerative diseases

doi: 10.1007/BF02471877

4.1 Revisiting classical pathways: context-dependent roles of Nrf2 and HDAC inhibition SFN activates the Nrf2 pathway through covalent modification of specific cysteine residues (Cys151, Cys273, and Cys288) on the Keap1 protein, leading to Nrf2 stabilization, nuclear translocation, and transcriptional activation of cytoprotective genes including NAD(P)H quinone oxidoreductase 1 (NQO1), heme oxygenase-1 (HO-1), and glutathione biosynthesis enzymes (63)

Restrictions: This test is not available in PA, AK, or HI

Primary indications for withdrawal of treatment were asthenia, worsening spasticity, and increases in serum aminotransferase levels and blood pressure

Our data demonstrate a significant increase in mitochondrial size in STZ-KETO compared to STZ-CHOW (Fig