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Changes in electrical activity of the heart along with an increase in sympathetic tone, catecholamines (epinephrine or adrenaline, norepinephrine or noradrenaline) and cortisol can contribute to the development of heart palpitations
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Possible mechanisms such as inflammation, oxidative stress, DNA damage, and protein misfolding by TMAO have been discussed in this review in the light of the latest advancements in the field
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