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CrossRef The clinical utilization of radiation therapy in Korea between 2009 and 2013 Jin-Kyu Kang, Mi-Sook Kim, Won-Il Jang, Young Seok Seo, Hee Jin Kim, Chul Koo Cho, Hyung Jun Yoo, Eun Kyung Paik, Yu Jin Cha, Hyun Jin Song Radiation Oncology Journal.2016

Single-cell analysis of skeletal muscle macrophages reveals age-associated functional subpopulations

Twelve patients diagnosed with GERD were given 500 ml of a liquid meal with identical protein and fat content but varying carbohydrate levels: one group received 848 g of carbohydrates, while the other received 1788 g

DNMT1 DNA Methyltransferase 1 DNMT3B DNA Methyltransferase 3B PRC2 Polycomb Repressive Complex 2 EZH2 Enhancer of Zeste 2 EED Embryonic Ectoderm Development IL-1b Interleukin-1 Beta IL6 Interleukin 6 EWAS Epigenome-Wide Association Study GPR56 G Protein-Coupled Receptor 56 5-aza-cytidine 5-Azacytidine RG108 A DNA Methyltransferase Inhibitor PD Parkinsons Disease SNCA Synuclein Alpha PARK7 Parkin 7 DJ-1 Parkinsonism Associated Deglycase SLC7A11 Solute Carrier Family 7 Member 11 HOTAIR HOX Transcript Antisense RNA SLC17A6 Solute Carrier Family 17 Member 6 PTPRN2 Protein Tyrosine Phosphatase Receptor Type N2 NR4A2 Nuclear Receptor Subfamily 4 Group A Member 2 PGC1-a Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha FXS Fragile X Syndrome FMR1 Fragile X Mental Retardation 1 CRISPR/Cas9 Clustered Regularly Interspaced Short Palindromic Repeats/Cas9 RRBS Reduced Representation Bisulfite Sequencing AP-1 Activator Protein 1 SOX2 SRY-Box Transcription Factor 2 5-hmC 5-Hydroxymethylcytosine YAC128 Yeast Artificial Chromosome 128 DhMRs Disease-Specific Hydroxymethylation Regions cfDNA Cell-Free DNA DMP Differentially Methylated Probes DMR Differentially Methylated Regions qPCR Quantitative Polymerase Chain Reaction Funding Statement The author(s) declare financial support was received for the research, authorship, and/or publication of this article

The term thrombophilia refers to familial or acquired hemostatic disorders that result in a predisposition to thromboembolic events.1 Inherited activated protein C resistance is the most frequent genetic cause of venous thrombosis.2 The factor V Leiden mutation is responsible for 95% of cases of activated protein C resistance.3 Activated protein C is a naturally occurring anticoagulant that is essential to the balance of procoagulant and anticoagulant mechanisms in the circulation.4 Protein C is activated by the binding of the thrombinthrombomodulin complex onto endothelial cells