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The ERO1-PDI axis is thought to be the main pathway for disulfide bond formation in ER (Wang et al., 2014)

however, they often ignore the chromophore/luminescent interactions with the probe, which can cause interferences in the results [151]

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Phospholipid release has been observed in experimental TBI models, which have shown an acute increase in serum SM and PC species as well as a decrease in serum TGs after mTBI in rats [86]

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