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At the mitochondrial level, these compounds regulate apoptotic signaling by increasing the expression of anti-apoptotic proteins such as B-cell lymphoma 2 (Bcl-2) and B-cell lymphoma-extra-large (Bcl-xL), while decreasing pro-apoptotic proteins including Bcl-2associated X protein (Bax) and Bcl-2associated agonist of cell death (Bad)

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doi: 10.1111/j.1365-2796.2011.02428.x 289 Cohen TervaertJWColarisMJvan der HulstRR

Urology 1994

In this context, NSAIDs, for example, are widely employed owing to their ability to inhibit cyclooxygenase (COX-1 and COX-2) enzymes