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We further found that knockdown of GGCT significantly reduced the GSH concentration in HCC and PCa cells and increased ROS production using DCFH-DA fluorescence detection (Fig

doi: 10.1038/nature08778

Although uptake via the MPC is the primary source of mitochondrial pyruvate, it is possible that other metabolic pathways can compensate when the MPC is compromised

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Notably, cysteinylglycine has a lower pKa and can more readily form thiolate that reduces Fe3+, potentially fueling Fenton chemistry and lipid oxidationillustrating context-dependent prooxidant effects of extracellular GSH catabolism