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These metabolomics findings are consistent with protection from liver steatosis and macrophage infiltration in fat depots in NQO1-Tg vs
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Prevention of salt-induced renal injury by activation of NAD(P)H:quinone oxidoreductase 1, associated with NADPH oxidase
Further research validated earlier results showing that decreased UA levels in MS represent a fundamental, intrinsic deficiency in protection against oxidative agents [158]
