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Description
It was well tolerated

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Thus, the transition from crystal deposition to acute inflammation can be conceptualized as a two-hit process: First hit: MSU crystal deposition on cartilage surfaces activates synovial fibroblasts to release CCL2/MCP-1, recruiting monocytes and establishing subclinical inflammation

Towards personalized allele-specific antisense oligonucleotide therapies for toxic gain-of-function neurodegenerative diseases
Think: natural killer cells (NK cells), a specialized lymphocyte T cell, that can detect and destroy invaders directly

10.1177/09645284241302816Online ahead of print
